Publications


FOSL1 inhibits type i interferon responses to malaria and viral infections by blocking TBK1 and TRAF3/ TRIF interactions;
Cai, B, Wu, J, Yu, X, Su, XZ & Wang, RF 2017, mBio, vol 8, no. 1, e02161-16. DOI:

A special issue on cancer immunotherapy;
Wang, RF 2017, Cell Research, vol 27, no. 1, pp. 1-2. DOI:

The TLR4–NOS1–AP1 signaling axis regulates macrophage polarization;
Srivastava, M, Saqib, U, Naim, A, Roy, A, Liu, D, Bhatnagar, D, Ravinder, R & Baig, MS 2016, Inflammation Research, pp. 1-12. DOI:

Cross-Regulation of Two Type I Interferon Signaling Pathways in Plasmacytoid Dendritic Cells Controls Anti-malaria Immunity and Host Mortality;
Yu, X, Cai, B, Wang, M, Tan, P, Ding, X, Wu, J, Li, J, Li, Q, Liu, P, Xing, C, Wang, HY, Su, XZ & Wang, RF 2016, Immunity, vol 45, no. 5, pp. 1093-1107. DOI:

Potential therapeutic targets for inflammation in toll-like receptor 4 (TLR4)-mediated signaling pathways;
Roy, A, Srivastava, M, Saqib, U, Liu, D, Faisal, SM, Sugathan, S, Bishnoi, S & Baig, MS 2016, International Immunopharmacology, vol 40, pp. 79-89. DOI:

USP38 Inhibits Type I Interferon Signaling by Editing TBK1 Ubiquitination through NLRP4 Signalosome;
Lin, M, Zhao, Z, Yang, Z, Meng, Q, Tan, P, Xie, W, Qin, Y, Wang, RF & Cui, J 2016, Molecular Cell, vol 64, no. 2, pp. 267-281. DOI:

TRIM14 Inhibits cGAS Degradation Mediated by Selective Autophagy Receptor p62 to Promote Innate Immune Responses;
Chen, M, Meng, Q, Qin, Y, Liang, P, Tan, P, He, L, Zhou, Y, Chen, Y, Huang, J, Wang, RF & Cui, J 2016, Molecular Cell, vol 64, no. 1, pp. 105-119. DOI:

Increased CD40 Expression Enhances Early STING-Mediated Type I Interferon Response and Host Survival in a Rodent Malaria Model;
Yao, X, Wu, J, Lin, M, Sun, W, He, X, Gowda, C, Bolland, S, Long, CA, Wang, R & Su, XZ 2016, PLoS Pathogens, vol 12, no. 10, e1005930. DOI:

TRIM11 Suppresses AIM2 Inflammasome by Degrading AIM2 via p62-Dependent Selective Autophagy;
Liu, T, Tang, Q, Liu, K, Xie, W, Liu, X, Wang, H, Wang, RF & Cui, J 2016, Cell Reports, vol 16, no. 7, pp. 1988-2002. DOI: